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In summary, metformin induces UCP2 overexpression in adipocytes, potentially minimizing oxidative stress due to drug-induced respiratory inhibition (Table 1)

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Since mitochondria cannot synthesize the negatively charged GSH, SLC25A39 is crucial for maintaining GSH homeostasis within the mitochondria [24]

Glutathione targets oxidative byproducts of fat metabolism, which is an entirely separate mechanism